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. 2024 Mar 8;10(10):eadl0165. doi: 10.1126/sciadv.adl0165

Table 1. Chemotherapies and targeted therapies inducing breast cancer dormancy or enabling dormancy escape in in vitro models.

Bcl-2, B-cell lymphoma 2 regulator protein; Oct-4, octamer-binding transcription factor 4; Sox-2 SRY-box transcription factor 2; G-CSF, granulocyte colony-stimulating factor; Tie2, tyrosine kinase 2 with immunoglobulin-like and epidermal growth factor homology domains.

Drug name Type of therapy Platform used Mechanism involved Effect on dormancy Reference
Docetaxel Chemotherapy SUM159 cells (TNBC) on 2D TCPS Induction (189)
Doxorubicin Chemotherapy SUM159 cells (TNBC) on 2D TCPS Induction (189)
Paclitaxel Chemotherapy MDA-MB-231-Br spheroids on 2D TCPS Low ERK/p38 activity ratio Induction (190)
Carboplatin Chemotherapy MDA-MB-231 on 2D TCPS and 3D polycaprolactone scaffolds Increase in cyclin D1, increase in Bcl-2, Oct-4, and Sox-2 Induction (191)
Docetaxel Chemotherapy D2.0R in 3D Matrigel with murine endothelial cells and embryonic fibroblasts Release of IL-6 and G-CSF Escape (35)
Tamoxifen Targeted therapy MCF7 on 2D TCPS OXPHOS down-regulation Induction (49)
Fulvestrant Targeted therapy MCF7 on 2D TCPS OXPHOS down-regulation Induction (49)
4-Hydroxytamoxifen Targeted therapy MCF7 in bone marrow endothelial niche on 3D Matrigel Endothelial Tie2 receptor expression and integrin β1 expression Escape (38)