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. 2024 Mar 13;24:59. doi: 10.1186/s12894-024-01444-5

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© The Author(s) 2024

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 1 — PFK-1 silencing inhibited BC progression. (A) The mRNA levels of PFK-1 were increased in SV-HUC-1, UM-UC-1, UM-UC-3, RT4, RT112, and T24 cells. UM-UC-1 and RT112 cells were used for next experiments. (B) After sh-PFK-1 transfection, the mRNA levels of PFK-1 in UM-UC-1 and RT112 cells was decreased. After sh-PFK-1 transfection, (C) CCK-8 assay indicated that cell viability of UM-UC-1 and RT112 cells was decreased. (D-E) Colony formation assay showed the cell growth of UM-UC-1 and RT112 cells were decreased. Transwell assay showed migration (F-G) and invasion (H-I) of UM-UC-1 and RT112 cells was decreased