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. 2024 Mar 16;15(3):220. doi: 10.1038/s41419-024-06592-y

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© The Author(s) 2024

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 1 — A The transcriptome expression of HMGA2 in RNA-seq datasets from 869 pancreatic cancer tissues and 417 adjacent normal tissues. B Proteomic expression of HMGA2 in PDAC samples from CPTAC patients. C The survival probability of patients with pancreatic cancer with high or low HMGA2 expression is shown as the median expression value of HMGA2. The data were collected from the TCGA database. D The UMAP plot shows the categorization of single cells from human pancreatic cancer cells into different cell types (left) and the single-cell expression levels of HMGA2 (right). E Reclustering of epithelial cells from D revealed that HMGA2 was highly expressed in the epithelial cluster 4 subtype. F Functional enrichment analysis of the marker genes of epithelial cluster 4 (log2FC > 3 and p value adjusted < 0.01) via the Metascape web server.