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. 2024 Mar 4;15:1344086. doi: 10.3389/fimmu.2024.1344086

Table 4.

Alternative therapeutical approaches to L-C19 sequelae.

Alternative treatment Results References
Cardiorespiratory and musculoskeletal fitness After 8 weeks intervention (3 session per week), muscle strength increased from 16% to 33%, and peak aerobic exercise capacity increased by 15%. (236)
6-min walk distance exercises During 6 weeks of testing, L-C19 patients presented increased pulmonary function, and improvements in dyspnea, fatigue, and quality of life. (237)
Sodium–glucose cotransporter 2
inhibitors and glucagon-like peptide 1 receptor agonists
Cardiovascular and renal benefits, improved metabolic function. (160)
Systemic corticosteroids After 6-8 weeks treatment, fatigue decreased by 41%, breathlessness by 46%, and cough by77%. (238)
Probiotics and short-chain fatty acids (SCFA) Restored gut microbiota and decreased GI disorders; improved mental health due to gut- brain axis modulation; SCFA maintained intact blood-brain barrier, downregulated microglial activation and reduced inflammation. (239, 240)
L-Arginine and liposomal Vitamin C 1.66 g of L-arginine and 500 mg liposomal Vitamin C for 28 days improved endothelial function, walking performance, reduced fatigue, and normalized pulmonary arterial hypertension. (241)
High-dose intravenous vitamin C 100-300 mg/kg body weight for 7 days improved oxygenation and reduced inflammation; ameliorated fatigue and associated symptoms such as sleep disorders, depression or pain. (242)
Vitamins (B group, C, D, E) and minerals (magnesium, zinc, selenium) Preventive effect on the development of autoimmunity, on alveolar damage, endothelial dysfunction, infection fervency reduction, balancing gut microbiota and coagulation. (243)
Pan-caspase inhibition Improved fatigue, dyspnea, cough, joint pain, anosmia, rheumatic fever. (244)
Acupuncture After 6 sessions, brain fog improved and joint pain reduced; blood stagnation reduced. (245)
Rosemary Carnosic Acid Neuroprotective and anti-inflammatory effects (limiting IL-1 β production by inhibition of NLRP3 inflammasome activation) (246)