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. 2024 Feb;86(1):1–15. doi: 10.18999/nagjms.86.1.1

Table 1.

Diseases classified as AiKDs, with their predisposing factors and mechanisms of pathogenesis (adopted and modified from References No. 17, 20, 22 and 26)

Disease Genetic causative or
predisposing factor
Pathogenetic mechanism
Pustular psoriasis and related conditions
Generalized pustular psoriasis (GPP) (including impetigo herpetiformis) 1) IL36RN loss-of-function variants

a) hyperactivation of the IL-36 pathway
2) CARD14 gain-of-function variants

b) upregulation of CARD14-NFκB signaling
3) AP1S3 loss-of-function variants

c) deficient AP1 endosomal translocation, defective autophagy
4) MPO loss-of-function variants

d) hyperactivation of the IL-36 pathway, defective efferocytosis of neutrophils
5) SERPINA3 loss-of-function variants

e) hyperactivation of the IL-36 pathway
6) BTN3A3 loss-of-function variant(s)

f) disturbed IL-1/IL-36 axis (?), upregulation of the TNF-α pathway (?)
acrodermatitis continua 1) – 4) above a) – d) above
palmoplantar pustular psoriasis (palmoplantar pustulosis) 1) – 3) above a) – c) above
Pityriasis rubra pilaris (PRP) (mainly type V) CARD14 gain-of-function variants
upregulation of CARD14-NFκB signaling
Porokeratosis loss-of-function variants in the mevalonate pathway-related genes MVK, MVD, PMVK and FDPS
disturbed mevalonate-isoprenylated protein pathway
Hidradenitis suppurativa 1) loss-of-function variants in the γ-secretase genes NCSTN, PSENEN and PSEN1
a) deficient γ-secretase, downregulated Notch signaling
2) variants in NOD2, LPIN2, NLRP3, NLRP12, PSMB8, MVK, IL1RN
b) other autoinflammatory pathways
Keratosis linearis with ichthyosis congenita and sclerosing keratoderma (KLICK) syndrome a single-nucleotide deletion in the 5′UTR of POMP (dysfunction of POMP)

proteasome maturation deficiency, ER stress and UPR
Familial keratosis lichenoides chronica NLRP1 gain-of-function variants (unknown)
hyperactivation of the NLRP1 inflammasome pathway
AiKD with EGFR deficiency EGFR loss-of-function variants
hyperactivation of PLA2, NFκB, and JNK1 signaling
AiKD with hepatitis and autism JAK1 gain-of-function variants
hyperactivation of JAK1-STAT signaling