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. Author manuscript; available in PMC: 2025 Apr 1.
Published in final edited form as: Arterioscler Thromb Vasc Biol. 2024 Feb 8;44(4):826–842. doi: 10.1161/ATVBAHA.123.319601

Figure 4.

Figure 4.

Acetylation-deficient p534KR increases cardiac glucose transporters and glycolytic function. A and B, Representative immunoblots and quantitative analysis of PFK-1, GLUT-1, GLUT-4, α-tubulin, and GAPDH in the indicated groups. PFK-1 and corresponding GAPDH were from the same membrane. Glut-1 and α-tubulin were from the same membrane. Glut-4 and corresponding GAPDH were from the same membrane. C, Cardiac F2,6-BP level was determined by the coupled-enzymatic assay and expressed as the fold change to the WT sham group. n=3–4. D, Cardiac PFK-1 activity was determined by the coupled-enzymatic assay and expressed as the OD340nm/min/mg protein. n=5–9.