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. 2024 Mar 28;16(1):2333463. doi: 10.1080/19490976.2024.2333463

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© 2024 The Author(s). Published with license by Taylor & Francis Group, LLC.

This is an Open Access article distributed under the terms of the Creative Commons Attribution-NonCommercial License (http://creativecommons.org/licenses/by-nc/4.0/), which permits unrestricted non-commercial use, distribution, and reproduction in any medium, provided the original work is properly cited. The terms on which this article has been published allow the posting of the Accepted Manuscript in a repository by the author(s) or with their consent.

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Figure 3. — Functional pathways required for the colonization in the oral and gut mucosae. The genes were grouped in functional categories and an SNR analysis was performed using input/output ratios of TnPKM sums (left). The heatmap (right) shows the transposon mutant gene abundance (log₁₀ averaged TnPKM, n = 4 to 6 mice) in the input (IN), the inflamed oral cavity (iOC), and the inflamed gut (iGT). The positive values of SNR indicate more in input. *, statistically significant TnPKM ratio (fold differences > 10, absolute SNR (|SNR|) > 5, p < 0.05, FDR < 0.05). AA, amino acid; TnPKM, transposon (tn) densities per kilobase per million mapped reads; SNR, signal-to-noise ratio; FDR, false discovery rate.