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. 2024 Mar 19;11:1346452. doi: 10.3389/fnut.2024.1346452

Table 2.

The correlation between iodine and PPARs.

Iodine and PPARs Object Mechanisms Year, references
PPARγ agonist (rosiglitazone) → RAI uptake↑ Thyroid carcinoma patients PPARγ expression increased in thyroid tissue 2008, (65)
I2↑ → PPARγ↑
I2↑ → PPARα↓
Human breast cancer cell line MCF-7 I2 treatment generates 6-IL derivative of AA, 6-IL binds specifically and with high affinity to PPARs 2009, (62)
I2↑ → PPARγ↑
I2↑ → PPARα↓
MUN induced mammary tumors in rats The presence of AA and formation of its 6-IL derivative in tumoral mammary gland 2009, (66)
I2↑or I↑ → PPARγ↑ DMBA induced mammary cancer in rats Prevent estrogen-induced DNA adducts through PPARγ/caspases pathways 2011, (67)
Iodine deprivation→PPARγ↑ Trophoblastic Cells Snail↑; MMP-9↑;
GCM-1↓; hGC↓; PAPP-A↓; E-cadherin↓;
2016, (68)
I2↑ → PPARγ↑ HeLa and SiHa cervical cancer cells; NOD/SCID mice CD49f, CK17, OCT-4, NANOG, SOX2 and KLF4↓ 2018, (69)
I2↑ → PPARγ↑ Women with early (stage II) and advanced (stage III) breast cancer 2019, (70)

PPARs, peroxisome proliferator-activated receptors; AA, arachidonic acid; 6-IL, 6-iodo-5-hydroxy-eicosatrienoic acid and the 6-iodolactone; RXRs, retinoic X receptors; RAI, radioiodine; MNU, methyl-nitrosourea; DMBA, dimethylbenz [a] anthracene; ROS, reactive oxygen species; MMP-9, matrix metalloproteinase-9; GCM-1, glial cell missing-1; PAPP-A, pregnancy-associated plasma protein-A.