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. 2024 Apr 1;24:402. doi: 10.1186/s12885-024-12182-0

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© The Author(s) 2024

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 6 — Knockdown of KCNJ11 does not affect cell growth and migration in MCF-7 cells. (A) Western blots of KCNJ11 and the indicated proteins in MCF-7 cells. (B) Cell viability measurement by CCK8. (C-D) Images of cell clones and EDU staining. (E) Images of wound healing assay to evaluate migration rate at 24 and 48 h. pLKO.1: the normal control cells; KCNJ11-sh1/2: KCNJ11 knockdown cells used KCNJ11 shRNA1 and KCNJ11-shRNA2