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editorial

. 2024 Mar 26;16(3):237–244. doi: 10.4252/wjsc.v16.i3.237

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©The Author(s) 2024. Published by Baishideng Publishing Group Inc. All rights reserved.

This article is an open-access article that was selected by an in-house editor and fully peer-reviewed by external reviewers. It is distributed in accordance with the Creative Commons Attribution NonCommercial (CC BY-NC 4.0) license, which permits others to distribute, remix, adapt, build upon this work non-commercially, and license their derivative works on different terms, provided the original work is properly cited and the use is non-commercial.

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Glucotoxicity in mesenchymal stem cells exposed to high glucose culture conditions and hyperglycemia. AGE: Advanced glycation end product; AMPK: AMP-activated protein kinase; CXCR: C-X-C chemokine receptor type 4; drp1: Dynamin-related protein 1; fis1: Fission protein 1. mfn1: Mitofusin 1; MSC: Mesenchymal stem cell; mTOR: Mammalian target of rapamycin; PI3K: Phosphoinositide 3-kinase; ROS: Reactive oxygen species; TNF: Tumor necrosis factor; TSC1: Tuberous sclerosis 1.