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Changing the balance of Th22‐Th17‐Treg cells is one of the crucial approaches to deteriorate and improve MAFLD. The imbalance of Th22‐Th17‐Treg cells homeostasis exacerbated inflammation and fibrosis, or even gut dysbacteriosis by proinflammatory related factors, but was reversed through the treatment of drugs and microRNA, especially traditional Chinese medicine, probiotics, gene editor, or adoptive cell transfer (ACT)
