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. 2024 Feb 15;14(2):e200266. doi: 10.1212/CPJ.0000000000200266

Table 2.

Clinical Outcomes

Pt. Etiology Outcome comments MRI Midbrain infarct LOS, dc Premorbid mRS 3-mo mRS 12-mo mRS
1 Small vessel Bilateral thalami Yes (Unilateral) 15 4 5 5
2 Cardioembolic Bilateral thalamic No 2 3 3 3
3 Small vessel Bilateral thalamic Yes (bilateral) 5 0 3 3
4 Hypercoagulability of malignancy EEG monitored, received Narcan Bilateral thalami, L parietal lobe, R cerebellar No 6 2 4 4
5 Small vessel Bilateral thalamus No 2 0 5 6
6 Cardioembolic Bilateral thalamic No 7 1 1
7 Small vessel Bilateral thalamic No 10 0 0 1
8 Cardioembolic Bilateral thalamic Yes (unilateral) 2 0 4 3
9 Small vessel Code Stroke (No tPA) Bilateral thalamus No 2 0 2
10 Hypercoagulability of malignancy Received Narcan Bilateral thalamic No 20 2 5 2
11 Large vessel atherosclerosis Bilateral thalamic Yes (bilateral) 21 0 4
12 Cardioembolic Received mannitol Bilat thalami, punctate embolic Yes (bilateral) 27 5 6 6
13 Large vessel atherosclerosis Code Stroke (No tPA) Bilateral thalamus No 2 4 4 4
14 Cardioembolic Stroke Code, Received tPA Bilateral thalamus No 7 4 3b 4
15 Small vessel Bilateral thalamus No 1 0a
Median (IQR) 6c (2.0–12.0) 1.5 (0–3.75) 4 (3.0–4.75) 4 (3.0–4.5)
a

Data not included in calculation of median because follow-up mRS was not available.

b

Patient had knee injury at baseline that improved at the 3-mo mark, reducing mRS at that time.

c

Length of stay rounded to the nearest whole days