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. 2023 Nov 27;28(1):2279813. doi: 10.1080/13510002.2023.2279813

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© 2023 The Author(s). Published by Informa UK Limited, trading as Taylor & Francis Group

This is an Open Access article distributed under the terms of the Creative Commons Attribution-NonCommercial License (http://creativecommons.org/licenses/by-nc/4.0/), which permits unrestricted non-commercial use, distribution, and reproduction in any medium, provided the original work is properly cited. The terms on which this article has been published allow the posting of the Accepted Manuscript in a repository by the author(s) or with their consent.

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Figure 5. — Hypoxia upregulates TGFβ1 level in WI38 fibroblast cells in vitro. (A) The mRNA level of TGFβ1 in WI38 cells under normoxia (NX) and hypoxia (HX) conditions for 24 h. (B–C) The protein level of TGFβ1 in cell body (B) and culture medium (C) of WI38 cells under normoxia (NX) and hypoxia (HX) conditions for 24 h. (D) The protein level of p-SMAD2 in WI38 cells under normoxia (NX) and hypoxia (HX) conditions for 24 h. n = 3 wells.