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. 2023 Nov 27;28(1):2279813. doi: 10.1080/13510002.2023.2279813

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© 2023 The Author(s). Published by Informa UK Limited, trading as Taylor & Francis Group

This is an Open Access article distributed under the terms of the Creative Commons Attribution-NonCommercial License (http://creativecommons.org/licenses/by-nc/4.0/), which permits unrestricted non-commercial use, distribution, and reproduction in any medium, provided the original work is properly cited. The terms on which this article has been published allow the posting of the Accepted Manuscript in a repository by the author(s) or with their consent.

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Figure 6. — Intermittent hypoxia promotes the progression and EMT process of lung cancer xenograft tumor in mice. (A) Tumor size of A549 xenograft tumor on mice in normal air (NA) or treated with intermittent hypoxia (IH). (B–F) The protein level of E-Cadherin (B and C), N-Cadherin (B and D), fibronectin (B and E), and p-SMAD2 (B and F) in tumor from mice in normal air (NA) or treated with intermittent hypoxia (IH) on day 20. n = 5 mice.