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. 2023 Dec 4;45(2):2285869. doi: 10.1080/0886022X.2023.2285869

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© 2023 The Author(s). Published by Informa UK Limited, trading as Taylor & Francis Group

This is an Open Access article distributed under the terms of the Creative Commons Attribution-NonCommercial License (http://creativecommons.org/licenses/by-nc/4.0/), which permits unrestricted non-commercial use, distribution, and reproduction in any medium, provided the original work is properly cited. The terms on which this article has been published allow the posting of the Accepted Manuscript in a repository by the author(s) or with their consent.

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Figure 3. — miR-192-5p directly targeted FTO by post-transcriptional mechanism. (A) Venn plot showed the interaction between the miR-192-5p targets and down-regulated mRNAs in GSE131454. (B) The predicted miR-192-5p binding site of FTO. (C) Dual-luciferase reporter assay was performed in HK-2 cells (n = 5). (D-E) FTO mRNA (D) and protein (E) expression regulated by miR-192-5p in HK-2 cells (n = 5). the data are represented as means ± SD, * p < .05, ** p < .01, compared with the agomir NC group.