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. 2024 Apr 8;7:427. doi: 10.1038/s42003-024-06111-x

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© The Author(s) 2024

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 7 — a UMAP plots of myeloid cell (Mye) (left). Proportion of Myeloid cell subclusters in 5 groups (right). b Dot plots of specific marker genes of Mye. c The age-associated changes of transcriptional noise of Mye subclusters. d. Gene ontology enrichment analysis of gene expression in cluster 4 of Mye (development). e Gene ontology enrichment analysis of gene expression in cluster 6 of Mye (aging). f Gene ontology enrichment analysis of up (top part, red)/down (bottom part, blue)-regulated gene expression in aging Mye. g Gene ontology enrichment analysis of gene expression in cluster 1 of tissue-resident macrophage (development). h Gene ontology enrichment analysis of gene expression in cluster 4 of tissue-resident macrophage (aging). i Gene ontology enrichment analysis of up (top part, red)/down (bottom part, blue)-regulated gene expression in aging tissue-resident macrophage.