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. 2024 Apr 8;14:8196. doi: 10.1038/s41598-024-58592-9

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© The Author(s) 2024

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Transcripts of RSAD2, CXCR3 and its ligands are more abundant in human carotid lesions where RSAD2 mRNA also correlates with CXCL10 and CXCL11 mRNA. The transcripts of (a) RSAD2, (b) CXCL9, (c) CXCL10, (d) CXCL11 and (e) CXCR3 in human atherosclerotic lesions (red circles) and adjacent intact tissues (blue circles) of matched patients with hypertension (n = 32) (GEO accession: GSE43292). Paired t-test followed by Benjamini and Hochberg corrections were employed to obtain adjusted p values and Pearson’s correlation was used to assess the relationship between RSAD2 mRNA and (f) CXCL9, (g) CXCL10 and (h) CXCL11 mRNA in human carotid lesions. Adjusted p values and Pearson’s correlation coefficients are indicated in the figures. p value < 0.05 is considered statistically significant. CXCR3, CXC chemokine receptor 3; CXCL9/10/11, CXC-motif chemokine 9/10/11; RSAD2, radical S-adenosyl-L-methionine domaine containing 2.