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. 2024 Mar 13;92(4):e00505-23. doi: 10.1128/iai.00505-23

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Fig 3 — DinJ inhibits the canonical inflammasome activation through Caspase-1. (A and C) Cytotoxicity of the BMDMs obtained from WT (A) and Casp1^-/- (C) C57BL/6 mouse. (B and D) ELISA analysis measuring the secretion of IL-1β in BMDMs obtained from WT (B) and Casp1^-/- (D) C57BL/6 mouse. (E) Western blotting analysis evaluating the cleavage of Caspase-1 and the expression levels of NLRP3 and NLRC4. β-actin served as the loading control. Statistical significance was determined at P values of < 0.01 (**) using the Student’s t-test.