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. 2024 Apr 11;16:55. doi: 10.1186/s13073-024-01327-2

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© The Author(s) 2024

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 4 — Gene expression dynamics in response to afatinib adaptation. A 549 driver genes clustered according to their expression. Cells are binned according to their reconstructed pseudotime. The average expression profile of the genes in the cluster and the cell composition of each bin as a percentage of cells in each timepoint is reported below each cluster. B Gene ontology enrichment analysis of 549 genes (bottom) and for each enriched term the percentage of genes belonging to a specific cluster of (A). C Genes in the enriched term EGFR tyrosine kinase inhibitor resistance and the cluster of (A) in which they fall