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. Author manuscript; available in PMC: 2025 Jan 31.
Published in final edited form as: FASEB J. 2024 Jan 31;38(2):e23404. doi: 10.1096/fj.202301158RR

TABLE 4.

ER stress induced by tunicamycin (TUNI) treatment led to decreased mitochondrial protein contents in control but not in deletion mice.

Gene Control Deletion


Protein Accession ID LFQ ratios TUNI/Vehicle p-Value TUNI/Vehicle LFQ ratios p-Value
ATP synthase subunit a P00848 Mtatp6 0.00 .062 1.47 .677
Cytochrome oxidase subunit 2 P00405 Mtco2 0.58 .018 1.23 .519
Ferrochelatase P22315 Fech 0.23 .009 0.88 .835
NADH–ubiquinone oxidoreductase chain 5 P03921 Mtnd5 0.62 .043 0.88 .460
NADH dehydrogenase flavoprotein 2 Q9D6J6 Ndufv2 0.74 .002 0.74 .159
Voltage-dependent anion-selective channel protein 2 Q60930 VDAC2 0.59 .033 0.75 .089
Voltage-dependent anion-selective channel protein 3 Q60931 VDAC3 0.53 .011 0.86 .440

Note: Data are expressed as LFQ ratio.