Skip to main content
. Author manuscript; available in PMC: 2025 Jan 31.
Published in final edited form as: FASEB J. 2024 Jan 31;38(2):e23404. doi: 10.1096/fj.202301158RR

TABLE 5.

ER stress induced by tunicamycin (TUNI) treatment led to an increase in select mitochondrial protein contents in control but not in deletion mice.

Gene Control Deletion


Protein Accession ID LFQ ratios TUNI/Vehicle p-Value TUNI/Vehicle LFQ ratios p-Value
ATPase inhibitor O35143 Atp5if1 5.87 .055 1.22 .827
ATP synthase subunit epsilon P56382 Atp5f1e 4.13 .072 1.12 .875

Note: Data are expressed as LFQ ratio.