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. 2024 Jan 10;24(1):69–80. doi: 10.1007/s40268-023-00449-z

Table 2.

Patient characteristics and demographics

Patient number 1 2 3 4 5 6 7 8 9 10
Age at start of study, years 1.2 9.8 5.7 2.3 2.3 5.5 3.8 4.5 12.8 9.5
Sex Male Female Male Male Female Female Male Male Male Male
Diagnosis MMA MMA MMA MMA PA PA PA PA PA PA
Gene affected MUT MUT MUT MUT PCCA PCCA PCCB PCCB NR PCCA
Genetic mutation Nonsense mutation Homozygous mutation Homozygous p. Tyr231* c.1420C>T Homozygous deletion of exons 23, 24 Homozygous deletion of exon 7 Homozygous p.Gly407Argfs*14 Homozygous c.1498 + 2T>C PCCA1 homozygous Homozygous p. Arg313*
Time of diagnosis Neonatal Neonatal Neonatal Neonatal Neonatal Neonatal Neonatal Neonatal Infant Neonatal
Complications at diagnosis
 Axial hypotonia
 Basal ganglia lesions
 Coma
 Encephalopathy
 Hypotonia
 Liver damage (jaundice)
 Metabolic acidosis
 Metabolic stroke
 Movement disorders
 Pancytopenia
 PDA
 Renal damage
 Respiratory distress
 RSV bronchiolitis
 Seizures
 Sepsis
 Vomiting
Complications after diagnosis
 Dystonia
 Intellectual disability

Arg arginine, C cytosine, Gly glycine, MMA methylmalonic aciduria, MUT methylmalonyl CoA mutase, NR not reported, PA propionic aciduria, PCCA propionyl-CoA carboxylase subunit alpha, PCCB propionyl-CoA carboxylase subunit beta, PDA small patent ductus arteriosus, RSV respiratory syncytial virus, T thymine, Tyr tyrosine

No patients were vitamin B12 responsive