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. 2024 Apr 9;15:1383494. doi: 10.3389/fneur.2024.1383494

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Copyright © 2024 Schmidt-Pogoda, Kaesmacher, Bonberg, Werring, Strecker, Koecke, Beuker, Gralla, Meier, Wiendl, Minnerup, Fischer and Minnerup.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Infarct growth after permanent large vessel occlusion. (A,B) Infarct growth after pMCAO in rodents without neuroprotective treatment. (A) Black lines connect the mean infarct volumes of untreated animals determined at timepoints 1 and 2 in each individual study. (B) Each cross represents the change in mean infarct volumes in an individual study. The median change in mean infarct volumes was 68%. (C,D) Infarct growth after pMCAO in rodents with neuroprotective treatment. (C) Black and green lines connect the mean infarct volumes of treated animals determined at timepoints 1 and 2 in each individual study. (D) Each cross represents the change in mean infarct volumes in an individual study. The median change in mean infarct volumes was 42%. (E,F) Infarct growth in stroke patients with TICI 0-2a thrombectomy. (E) Black lines connect the infarct volumes at timepoints 1 and 2 in each patient. The red line represents an overall regression line given by the fixed effects of the mixed model. (B) Each cross represents the infarct volume growth in a patient. The median change in infarct volumes was 148%.