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. 2024 Apr 25;15(4):e00686. doi: 10.14309/ctg.0000000000000686

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© 2024 The Author(s). Published by Wolters Kluwer Health, Inc. on behalf of The American College of Gastroenterology

This is an open access article distributed under the terms of the Creative Commons Attribution-Non Commercial-No Derivatives License 4.0 (CCBY-NC-ND), where it is permissible to download and share the work provided it is properly cited. The work cannot be changed in any way or used commercially without permission from the journal.

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Figure 1. — NGAL expression increases in the plasma of subjects with CP. (a) NGAL measured by ELISA from the multicenter (PROCEED) study discovery set. Statistical significance was determined by a non-parametric 1-way ANOVA with Tukey's multiple comparisons correction. (b) AP/RAP and CP samples were subdivided by diabetes status and compared for plasma NGAL concentration. Statistical significance was determined by 2-way ANOVA with Tukey's multiple testing correction. (c) ROCs with AUCs of plasma NGAL comparing control vs CP and (d) AP/RAP vs CP. ***P < 0.001, ****P < 0.0001. AP, acute pancreatitis; CP, chronic pancreatitis; ELISA, enzyme-linked immunosorbent assay; NGAL, neutrophil gelatinase-associated lipocalin; RAP, recurrent acute pancreatitis.