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. 2024 Mar 7;90(4):101411. doi: 10.1016/j.bjorl.2024.101411

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© 2024 Published by Elsevier España, S.L.U. on behalf of Associação Brasileira de Otorrinolaringologia e Cirurgia Cérvico-Facial.

This is an open access article under the CC BY license (http://creativecommons.org/licenses/by/4.0/).

PMC Copyright notice

EPHX4 promoted the proliferation, migration of laryngeal cancer cell line. (A) Immunoblotting of normal control (NC) versus EPHX4 knowdown with sh#1, sh#2, and vector versus EPHX4-overexpressing (EPHX4) efficiencies in HN6 cell line. (B) CCK-8 assay was performed in NC and EPHX4 knockdown and overexpression in HN6 cell line. (C) Clonogenic assays of NC and EPHX4 knockdown and overexpression HN6 cell line were recorded (left) and quantitatively analyzed (right). (D) Wound healing assays of EPHX4 knockdown and overexpression in HN6 cell line were recorded. (E) Left: Representative IHC images of EPHX4 protein levels in LSCC tumors and in paired normal tissues.Scale bar:100 uM. Right: EPHX4 protein levels were significantly lower in ESCC tumors than in paired normal tissues (n = 12).