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. Author manuscript; available in PMC: 2024 May 1.
Published in final edited form as: J Alzheimers Dis. 2020;78(4):1765–1774. doi: 10.3233/JAD-200992

Table 3.

Comparison of the low and high CAA-SVD score groups in the mixed CMBs group with a high HA-SVD score

low CAA-SVD score (n = 11) high CAA-SVD score (n = 32) p
Age, y, mean±SD 81.2±6.1 74.8±8.4 0.025
Male, n (%) 4 (36.4) 14 (43.8) 0.736
MMSE, median (IQR) 20 (18–23) 24 (19–26) 0.356
Hypertension, n (%) 5 (45.5) 14 (43.8) 1
Diabetes mellitus, n (%) 2 (18.2) 5 (15.6) 1
Dyslipidemia, n (%) 7 (38.9) 15 (42.9) 1
Smoking, n (%) 3 (27.3) 7 (21.9) 0.698
HA-SVD-Score, median (IQR)
 Lacune 0 (0–1) 1 (1–1) 0.483
 CMBs 1 (1–1) 1 (1–1) 1
 BG-PVS 1 (1–1) 1 (1–1) 0.880
 WMH 1 (0–1) 1 (1–1) 0.386
 Total 3 (3–4) 4 (3–4) 0.154
CAA-SVD-Score, median (IQR)
 Lobar CMBs 0 (0–1) 2 (2–2) <0.001
 cSS 0 (0–0) 0 (0–0) 0.555
 CSO-PVS 0 (0–1) 1 (0–1) 0.117
 WMH 1 (0–1) 1 (1–1) 0.386
 Total 2 (1–2) 4 (3–4) <0.001

BG, basal ganglia; CAA, cerebral amyloid angiopathy; CMB, cerebral microbleed; CSO, centrum semiovale; cSS, cortical superficial siderosis; HA, hypertensive arteriopathy; IQR, interquartile range; MMSE, Mini-Mental State Examination; PVS, perivascular space; SD, standard deviation; SVD, small vessel disease; WMH, white matter hyperintensity.

p < 0.05 for low CAA-SVD score versus high CAA-SVD score.