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. 2024 May 2;19:34. doi: 10.1186/s13062-024-00468-z

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© The Author(s) 2024

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 9 — Knockdown PCED1B-AS1 inhibited the progression of gastric cancer in mice. A The histopathological changes of gastric cancer were detected by H&E staining. Immunohistochemical staining was used to detect the expression of MAP2K7 (B), E-cadherin (C), N-cadherin (D) and Vimentin (E) in gastric cancer tissues. F Representative tumor tissue. G Representative nude mice are shown. H Statistical analysis of tumor volume (H) and tumor weight (I) from tumors obtained from two groups. The data were presented as mean ± SEM, versus si-NC group, *P < 0.05, **P < 0.01, n = 6