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The mechanics underlying left atrial vortex flow in patients with postoperative congenital heart disease (CHD) are demonstrated. In such individuals, surgical intervention leads to the development of fibrosis and remodeling, which subsequently results in augmented left atrial stiffness, pressure, and volume loading. It is hypothesized that this heightened stiffness diminishes left atrial reservoir function, ultimately leading to the disappearance of left atrial vortex flow
