Table 1.
Dominant Microbiota in Humans and Mice | |||
---|---|---|---|
Authors | Dominant microbiota | Host | |
Li et al. [43] | Lactobacillus reuteri, Enterococcus faecium, Escherichia coli, Bacteroides ovatus, Fusobacterium gastrosuis | Mice | |
Rinninella et al. [44] | Fecalibacterium prausnitzii, Clostridium spp., Lactobacillus reuteri, Enterococcus faecium, Bacteroides vulgatus, Bacteroides uniformis Prevotella spp., Parabacteroides distasonis | Human | |
Predominantly Beneficial Microbiota in Various Pathologies | |||
Authors | Micro-organism | Pathology | Main Findings |
Lima et al. [45] | Odoribacter splanchnicus | Ulcerative Colitis | Odoribacter splanchnicus plays a crucial role in enhancing both metabolic functions and immune cell resilience against colitis. |
Yang et al. [46] | Lactobacillus acidophilus, coleohominis, gallinarum; Selenomonas artemidis | Constipation, depression, and anxiety | Psychiatric symptoms were improved after the FMT. |
Aggarwala et al. [47] | Bacteroides vulgatus, uniformis ovatus, cellulosilyticus; Parabacteroides distasonis, merdae |
Clostridioides difficile infection |
Significantly predicted the clinical outcomes of the transplantation for up to five years. |
Lee et al. [48] | Bacteroidales | Non specific intestinal disorders | The study effectively used genome-resolved metagenomics to track and identify bacterial strains that persist in FMT recipients, deepening insights into microbiota dynamics post-transplant. |
Zhang et al. [49] | Faecalibacterium; Eubacterium; Roseburia | Inflammatory Bowel Disease | Symptoms were improved after FMT but outcomes are linked to gut microbiota and methodology variations, emphasizing the need for standardized research to improve FMT effectiveness through microbial and metabolite adjustments. |