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. 2024 May 13;81(1):215. doi: 10.1007/s00018-024-05245-9

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© The Author(s) 2024

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 5 — Treatment with sPIF restores neurogenesis in Dp(16)1Yey mice pups. Representative images of immunostaining of NeuN (red) and BrdU (green) in the dentate gyrus of WT pups (vehicle: n = 8; sPIF: n = 7) and Dp(16)1Yey pups (vehicle n = 6; sPIF: n = 7) (A). Quantification of the number of NeuN-and BrdU-positive cells in the dentate gyrus, normalized against the surface area (A). Data are expressed as the mean ± SD and were analyzed in a two-way ANOVA. An interaction between genotype and treatment was not found, and pairs of groups were compared in Student’s t-test. ** p < 0.01