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. 2024 May 14;14(1):81. doi: 10.1038/s41408-024-01066-y

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© The Author(s) 2024

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 1 — A Relationship between NUP98 fusions and concurrent mutations. Top 10 mutations were shown. B Co-occurrence and mutual exclusion analysis. Only top 20 mutation and NUP98 fusions were shown. Both (A) and (B) demonstrated NUP98::NSD1 and FLT3-ITD were the mostly concurrent gene abnormalities. C Heatmap depicting the immunophenotypic landscape of patients. Positive markers were shown with red, partial positives were shown with pink, cross-lineage antigen positives were shown with blue and negative markers were shown with green.