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. 2024 May 20;14:11497. doi: 10.1038/s41598-024-62262-1

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© The Author(s) 2024, corrected publication 2024

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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PDK4 upregulation in TAZ-KO cells exacerbates AMPK activation and FOXO1 expression. Phosphorylation of AMPK residue Thr¹⁷² (p-AMPK) and FOXO1 protein levels were assayed in WT and TAZ-KO myoblasts treated with PDK4-targeted siRNA. The data points represent means ± S.D. (error bars) for each individual biological replicate of each group. * 0.05 < p < 0.1, *** 0.001 < p < 0.01.