Fig. 6.

Model for Pelo-Hbs1-mediated TE silencing at translational level. The transposon transcripts have escaped the transcriptional and post-transcriptional gene silencing and have loaded to ribosomes for translation, and could be degraded by the Pelo-Hbs1 complex via an NGD-like mechanism. During the ribosome elongation, some unknown factors, possibly piRNAs, could cause ribosome stall, followed by Pelo-Hbs1 recruitment and mRNA decay