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. 2024 Apr 25;4(5):625–637. doi: 10.1038/s43587-024-00626-y

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© The Author(s) 2024

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Extended Data Fig. 6 — In unimpaired participants (EC, N = 65; Hippocampus, N = 66; IT, N = 66; LC, N = 66 independent individuals represented by dots), LC tangle density is related to tangles in MTL structures, such as the hippocampus (upper plot) and the entorhinal cortex (middle plot), and the inferior temporal cortex (lower plot). These associations are consistent with the pattern observed in impaired (EC, N = 93; Hippocampus, N = 94; IT, N = 94; LC, N = 94 independent individuals with mild cognitive impairment or AD; represented by triangles) participants. Lines in the scatter plots represent linear trends. LC tangle density histogram appears on the upper part of the plots, while hippocampus, entorhinal cortex, or inferior temporal cortex tangle density histograms appear on the right-side of the respective plots.