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. 2024 May 23;44(6):133. doi: 10.1007/s10875-024-01726-5

Table 2.

The clinical, immunological and genetic phenotype of the CVID cohorts

Phenotypes CVID_IEL
(n=5)
CVID_N
(n=3)
CVID total
(n=8)
Clinical
Infection only, n 0 0 0%a
Complicationsb 5 3 100% a
Enteropathy 5 0 63% a
Splenomegaly 2 2 50% a
Lymphoid hyperplasia 1 2 38% a
Organ-specific autoimmunity 2 0 25% a
Autoimmune cytopenia 1 1 25% a
IgG replacement therapy 5 3 100% a
Immunomodulatory treatment 0 0 0% a
Immunological
Ig at diagnosis
Reduced IgA (< 0.7 g/L) 5 3 100% a
Reduced IgM (< 0.4 g/L) 4 3 88% a
IgG, g/Lc 2.5 (1.2–3.9) 1.7 (0.5–3.0) 2.5 (0.5–3.9)
B cell sub-classesd
CD19+ cells of lymphocytes> 1% 5 3 100% a
Switched memory B-cells2% 4 2 75% a
Transitional B cells <9 % 3 3 75% a
CD21low B cells ≥ 10% 3 3 75% a
Total B cells (× 106/L) c 72 (31–306) 349 (137–579) 218 (31–579)
Total T cells (× 106/L) c 824 (471–1404) 1730 (1131–3483) 1246 (471–3483)
CD4+ T cells (× 106/L) c 496 (170–879) 803 (647–1054) 601 (170–1054)
CD8+ T cells (× 106/L) c 426 (303–805) 1008 (301–2429) 430 (301–2429)
NK cells (× 106/L) c 147 (28–362) 129 (101–531) 138 (28–531)
Genetics
Known monogenic PID mutation e 1 0 13% a

aPercentage of the total CVID Cohort (n=8) that have the phenotype in the first column

bNone of the patients had granulomas, lymphoid interstitial pneumonitis, nodular regenerative hyperplasia of the liver or lymphoma

cMedian (minimum-maximum)

dB cells are classified according to EURO classification [29]

eWhole exome sequencing and then gene panel (covering 598 genes, see methods)

CVID, Common variable immunodeficiency; CVID_IEL, Subclassification of duodenal biopsies from the CVID patient with increased intra epithelial lymphocytes; CVID_N, Subclassification of duodenal biopsies from the CVID patient with no increased intra epithelial lymphocytes (normal); Ig, Immunoglobulin; NK, natural killer cells; PID, primary immunodeficiency