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. 2023 Dec 9;14(5):100917. doi: 10.1016/j.jpha.2023.12.007

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© 2023 The Author(s)

This is an open access article under the CC BY-NC-ND license (http://creativecommons.org/licenses/by-nc-nd/4.0/).

PMC Copyright notice

Fig. 2 — Myocardial metabolism in atrial fibrillation (AF). Glucose metabolism was significantly enhanced to compensate for fatty acid (FA) oxidation. Mitochondrial function matters in many pathophysiological processes of AF. FABP: fatty acid-binding protein; GLUT: glucose transporter; MCT: monocarboxylate transporter; ADP: adenosine diphosphate; ATP: adenosine triphosphate; NADH: nicotinamide adenine dinucleotide; NAD: nicotinamide adenine dinucleotide; ANT: adenine nucleotide translocator; CPT: carnitine palmitoyl transferase; MPC: mitochondrial pyruvate carrier; OAA: oxaloacetic acid; TCA: tricarboxylic acid cycle; α-KG: α-ketoglutaric acid.