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. 2024 May 31;10:267. doi: 10.1038/s41420-024-02039-7

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© The Author(s) 2024

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 8 — This diagram illustrates the mechanistic pathway by which bleomycin induces apoptosis in cervical cancer cells. It demonstrates the involvement of Peroxiredoxin I and II in modulating the levels of reactive oxygen species (ROS) and their impact on endoplasmic reticulum (ER) stress, subsequently leading to cell apoptosis. The diagram also highlights the role of the PI3K/AKT signaling pathway in this process.