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. 2024 May 15;27(6):109979. doi: 10.1016/j.isci.2024.109979

Table 2.

Micro-RNAs (miRNAs) in cancer associated drug resistance

miRNA Target Chemotherapy Drug Correlation
miR-646 CDK6 pathway Cisplatin hsa_circ_0081143 contributes to cisplatin resistance in gastric cancer by targeting the miR-646/CDK6 pathway.
miR-451 YWHAZ Paclitaxel miR-451 plays a role in paclitaxel resistance by regulating YWHAZ in breast cancer.
miR-198 PIK3R1 Cisplatin circAKT3 contributes to cisplatin resistance in gastric cancer by boosting PIK3R1 expression through miR-198 suppression.
miR-155 N/A Cisplatin The delivery of miR-155 through exosomes contributes to the development of cisplatin resistance in oral cancer cells by promoting EMT.
miR-19b mRNAs 5-fluorouracil miR-19b is upregulated in response to 5-fluorouracil (5-FU), and it influences the cell cycle by targeting specific mRNAs.
miR-208b PDCD4 Oxaliplatin Exosomal miR-208b contributes to oxaliplatin resistance in colorectal cancer by promoting Treg expansion through targeting PDCD4.
miR-17-5p Beclin1 Paclitaxel miR-17-5p downregulation is associated with paclitaxel resistance in lung cancer. It achieves this by directly altering Beclin1 expression, a crucial modulator of autophagy.
miR-873 CDK3 Tamoxifen miR-873 acts as a tumor suppressor in ER-positive breast cancer by reducing ERα transcriptional activity and overcoming tamoxifen resistance through CDK3 targeting.
miR-449a ADAM22 Tamoxifen miR-449a combats tamoxifen resistance in human breast cancer cells by targeting ADAM22, a key player in this process.
miR-34a-5p and miR-204-5p ACSL4 Docetaxel LncRNA NEAT1 contributes to docetaxel resistance in prostate cancer by upregulating ACSL4 through miR-34a-5p and miR-204-5p interactions.
miR-375 N/A Paclitaxel miR-375 exhibits increased expression in cervical cancer cells that have developed paclitaxel resistance.
miR-186 CPEB2 Methotrexate TUG1 contributes to methotrexate resistance in colorectal cancer by modulating the miR-186/CPEB2 axis.
miR-200c and miR-141 N/A Oxaliplatin Down-regulation of miR-200c and miR-141 contributes to selective resistance to oxaliplatin and EMT in colorectal cancer cells during repeated treatments with L-OHP.
miR-130b Colony-stimulating factor 1 Cisplatin, Paclitaxel miR-130b is epigenetically silenced in ovarian cancer, leading to the development of multidrug resistance by targeting colony-stimulating factor 1.
let-7 Hedgehog signaling Erlotinib/Cisplatin Blocking Hedgehog signaling enhances the sensitivity of NSCLC cells to conventional therapies by influencing the expression of miRNAs that regulate EMT.
miR-21 PTEN/PI3K/Akt pathway Paclitaxel miR-21 overexpression associated with the suppression of PTEN, activating the PI3K/Akt pathway, and promoting resistance to Paclitaxel in various cancers.
miR-203 ATM kinase Oxaliplatin miR-203 contributes to oxaliplatin resistance in colorectal cancer cells by negatively regulating ATM kinase.
miR-200b FN1 Doxorubicin miR-200b modulates EMT in chemo-resistant breast cancer cells by directly targeting FN1.
miR-21 PTEN Trastuzumab miR-21 overexpression contributes to trastuzumab resistance in HER2+ breast cancers.
miR-22 PTEN Tamoxifen CD63+ cancer-associated fibroblasts (CAFs) promote tamoxifen resistance in breast cancer cells by secreting exosomes rich in miR-22.
miR-374a-5p N/A Oxaliplatin miR-374a-5p, elevated in patients with gastric cancer, predicts therapy response. It induces drug resistance by targeting Neurod1. Exosome-delivered anti-miR-374a-5p restores sensitivity to oxaliplatin in gastric cancer cells.
MiR-99a and MiR-491 CAPNS1 Cisplatin MiR-99a and MiR-491 play a crucial role in regulating cisplatin resistance in human gastric cancer cells.
miR-196a CDKN1B and ING5 Cisplatin Exosomal miR-196a, originating from cancer-associated fibroblasts (CAFs), induces cisplatin resistance in head and neck cancer (HNC).
miR-29 ERK 1/2 Cisplatin miR-29 downregulation is associated with cisplatin resistance in ovarian cancer cells.
MiR-148a MSK1 paclitaxel MiR-148a plays a crucial role in reducing paclitaxel resistance in hormone-refractory, drug-resistant prostate cancer cells.
miR-100 and miR-125b Wnt/β-catenin signaling pathway Cetuximab lncRNA MIR100HG, along with its embedded microRNAs miR-100 and miR-125b, contributes to cetuximab resistance by modulating the Wnt/β-catenin signaling pathway.
miR-192 DNA damage response pathways Doxorubicin miR-192 downregulation linked to impaired DNA damage response, contributing to resistance to Doxorubicin in various cancers.
miR-19a N/A FOLFOX Serum miR-19a serves as a potential molecular biomarker for predicting and monitoring resistance to first-line FOLFOX chemotherapy regimens in advanced patients with colorectal cancer.
miR-224 N/A Methotrexate miR-224 is underexpressed in methotrexate-resistant human colon cancer cells.
miR-153 FOXO3a Platinum miR-153 contributes to colorectal cancer progression by enhancing invasion and promoting chemotherapeutic resistance.
miR-200c EMT pathway Gemcitabine miR-200c downregulation associated with EMT and resistance to Gemcitabine in pancreatic cancer.
miR-128 ABCC5 and Bmi-1 Cisplatin miR-128 plays a role in promoting cisplatin resistance in ovarian cancer by reducing ABCC5 and Bmi-1 expression.
miR-193a-3p LOXL4 Pirarubicin, Paclitaxel, Adriamycin, Epirubicin Hydrochloride, Cisplatin miR-193a-3p influences bladder cancer chemoresistance by directly targeting the LOXL4 gene and affecting the Oxidative Stress pathway.
miR-320c SMARCC1 Gemcitabine miR-320c regulates gemcitabine resistance in pancreatic cancer by targeting SMARCC1, a core subunit of the chromatin remodeling complex.
miR-381 MDR1 Cisplatin miR-381 combats cisplatin resistance in breast cancer by targeting MDR1, a key player in drug resistance.
miR-503 Bcl-2 Cisplatin miR-503 modulates cisplatin resistance in non-small cell lung cancer cells by targeting Bcl-2.
miR-29b N/A Tamoxifen miR-29b downregulation contributing in resistance to Tamoxifen in breast cancer.
miR-17-92 TGF-β1 Gemcitabine The miR-17-92 cluster opposes the state of dormancy and resistance to chemotherapy in a specific subset of pancreatic cancer stem cells.
miR-619-5p Wnt/β-catenin and autophagy pathway Gemcitabine LncRNA PVT1 contributes to gemcitabine resistance in pancreatic cancer by activating the Wnt/β-catenin and autophagy pathway through the modulation of the miR-619-5p/Pygo2 and miR-619-5p/ATG14 axes.
miR-634 Ras-MAPK pathway Cisplatin miR-634 enhances drug sensitivity in resistant ovarian cancer cells by directly targeting the Ras-MAPK pathway.
miR-217 N/A Paclitaxel chemotherapy-induced exosomal circBACH1 contributes to breast cancer resistance and stemness by interacting with miR-217 and upregulating the expression of G3BP2.
MiR-30a AKT Gemcitabine MiR-30a modulates the responsiveness of cancer cells to chemotherapy by influencing the SNAI1/IRS1/AKT pathway.
miR-15b and miR-16 BCL2 Vincristine, Adriamycin, 5-fluorouracil, cisplatin, mitomycin C, etoposide miR-15b and miR-16 regulate multidrug resistance in human gastric cancer cells by targeting BCL2.
miR-125a-3p N/A Gemcitabine In PDAC, miR-125a-3p governs chemosensitivity by suppressing epithelial-mesenchymal transition through the inhibition of Fyn.
miR-153 N/A Gemcitabine miR-153 augments the therapeutic efficacy of gemcitabine through the targeting of Snail in the context of pancreatic cancer.
miR-183 N/A Gemcitabine A newly identified interplay between KLF4 and ZEB1 governs the resistance to gemcitabine in pancreatic ductal adenocarcinoma.
miR-200 N/A Gemcitabine Elevation of miR-200 and let-7 expression induced by natural agents results in the reversal of epithelial-to-mesenchymal transition in pancreatic cancer cells resistant to gemcitabine.
MiR-130a and MiR-374a PTEN Cisplatin MiR-130a and MiR-374a Serve as New Regulators of Cisplatin Resistance in A2780 Human Ovarian Cancer Cells.
miR-1252 N/A Paclitaxel circCELSR1 (hsa_circ_0063809) plays a role in paclitaxel resistance of ovarian cancer cells by regulating FOXR2 expression via miR-1252.
miR-205 N/A Gemcitabine Induction of epithelial-to-mesenchymal transition by macrophage migration inhibitory factor amplifies tumor aggressiveness and serves as a prognostic predictor in surgically resected pancreatic ductal adenocarcinoma.
miR-214 PTEN Cisplatin miR-214 promotes cell survival and confers resistance to cisplatin by directly targeting PTEN.
miR-222 and miR-29a N/A Adriamycin and docetaxel miR-222 and miR-29a play a role in fostering drug resistance in breast cancer cells.
miR-205 and miR-31 BCL2L2 (encoding Bcl-w) and E2F6 Docetaxel, Cisplatin Reduced expression of miR-205 and miR-31 leads to resistance against apoptosis induced by chemotherapy in prostate cancer cells.
miR-497 NF-κB1 Gemcitabine microRNA-497 averts gemcitabine resistance, migration, and invasion in pancreatic cancer stem cells by directly targeting nuclear factor kappa B-1.
MiR-487a BCRP/ABCG2 Mitoxantrone MiR-487a reverses mitoxantrone (MX) resistance in breast cancer cells by targeting the breast cancer resistance protein (BCRP/ABCG2).
miR-506 N/A Gemcitabine Reduced expression of NEAT1 heightens the sensitivity of gemcitabine-resistant pancreatic cancer cells to gemcitabine via the modulation of the miR-506-3p/ZEB2/EMT axis.
miR-106a and miR-591 N/A Paclitaxel miR-106a and miR-591 play crucial roles in causing paclitaxel resistance in ovarian cancer cells.
miR-26a and miR-30b CCNE2 Trastuzumab miR-26a and miR-30b are key players in HER2+ breast cancer trastuzumab resistance, with their target gene being CCNE2.
miR-509 N/A Gemcitabine miR-509-5p and miR-1243 enhance gemcitabine sensitivity by suppressing epithelial-mesenchymal transition in pancreatic cancer.
miR-27a N/A Vincristine, Adriamycin, Cisplatin, 5-fludrouracil Reducing miR-27a levels could potentially hinder the growth and enhance sensitivity to drugs in gastric cancer cells.
miR-1206 N/A Gemcitabine Circular RNA circ_0092367 suppresses epithelial-mesenchymal transition (EMT) and mitigates gemcitabine resistance in pancreatic cancer through the regulation of the miR-1206/ESRP1 axis.
miR-509 N/A Gemcitabine miR-509-5p and miR-1243 enhance gemcitabine sensitivity in pancreatic cancer by impeding epithelial-mesenchymal transition.