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. 2024 Jun 4;15(6):392. doi: 10.1038/s41419-024-06706-6

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© The Author(s) 2024

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 1 — a Sample correlation analysis of the transcriptome of human epidermal equivalents from the control group, UVB_24 h group and UVB_72 h group. b Principal component analysis of the transcriptome of human epidermal equivalents from the control group, UVB_24 h group and UVB_72 h group. c Clustering heatmap of the 1227 differentially expressed genes based on FPKM. d, e Over-representation analysis of GO (d) and KEGG pathways (e) based on the 1227 differentially expressed genes. The size indicates the gene numbers and the color corresponds to the P value. f Gene set enrichment analysis (GSEA) of differentially expressed genes in the UVB_24 h group relative to the control group, UVB_72 h group relative to the control group and UVB_72 h group relative to the UVB_24 h group. g Expression heatmap of the top 10 changed genes.