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Table 5.

Pathological and phenotypic characteristics in 15 Helicobacter pylori‐ and atrophy‐negative patients

Pathology T factor MUC5AC MUC6 MUC2 Villin Cdx2 Phenotype in parts §
Case 1 sig se +++ ++ ± ++ G type
Case 2 por sm ++ + + + GI type
Case 3 sig mp ++ ± G type
Case 4 sig si + ± G type
Case 5 por OP (–)
Case 6 tub2 ss ± ± ± +++ N type
Case 7 tub1 m N type
Case 8 tub2 OP (–)
Case 9 sig sm ++ ++ ± +++ GI type
Case 10 muc ss ++ + ++ ++ GI type
Case 11 sig sm ++ + + ++ GI type
Case 12 muc sm ++ ++ ++ + I type
Case 13 tub2 m ++ ++ ++ +++ GI type
Case 14 tub2 ss ± + + +++ I type
Case 15 sig si + ± ± G type

tub1 and tub2 indicate well differentiated‐type and por, muc and sig indicate poorly differentiated‐type gastric cancer according to the Japanese Classification of Gastric Carcinoma.

Depth of invasion indicate mucosa (m), submucosa (sm), muscularis propria (mp), subserosa (ss), serosa‐exposed (se), si (serosa‐infiltrating) and OP (–) indicating no operation.

§

+Cases with more than 10% of the section area consisting of at least one gastric or intestinal epithelial cell phenotype were classified as gastric (G type) or intestinal (I type) phenotype cancer, respectively. Other cases were classified as either the gastric or intestinal mixed phenotype (GI type), while those with no expression phenotype were grouped as unclassified (N type).