Figure 1.

(A) Sixteen hours after the onset of typical symptoms, a fluid-attenuated inversion recovery (FLAIR) MRI sequence revealed abnormal subcortical and cortical hyperintensities consistent with vasogenic edema in the bilateral temporal, parietal, and occipital lobes, with more prominent in the left hemisphere (red arrows). Apparent diffusion coefficient (ADC) mapping demonstrates cortical/subcortical vasogenic edema with hyperintensity (yellow arrow). Diffusion-weighted imaging (DWI) showed isointensity in these areas. (B) Twenty-two hours after the onset of typical symptoms, the electroencephalography demonstrated mild slowing of the background rhythm, left dominant slowing of the posterior background activity and posteriorly left dominant bilateral PLEDs plus fast activity.