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. 2024 Feb 2;29(5):1440–1449. doi: 10.1038/s41380-024-02422-x

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© The Author(s) 2024

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 5 — a CHGB-derived neuropeptides present in both SZ and control iNs. Neuropeptides derived from the CHGB proneuropeptide were identified and quantitated in KCl secretion media of both SZ and control iNs. Relative neuropeptide levels are indicated as peak intensity area from LC-MS/MS. Neuroepeptide levels for each condition are shown as means ± s.e.m (n = 7 for the SZ group, and n = 6 for the control group). b CHGB-derived neuropeptides present in only control iNs. CHGB neuropeptides observed in only the control iN group were quantitated as described in the methods. Data are shown as the mean ± s.e.m. (n = 6 iN control cell lines).