Yao 2006.

Methods	Study design: randomised controlled trial. Method of randomisation: no description. Concealment of allocation: no description. Exclusions post randomisation: none. Length of follow‐up: 6 months. Losses to follow‐up: none. Intention‐to‐treat analysis: not mentioned.
Participants	Participants were patients with CHD, with evidence of coronary arteriography showing stenosis ≥ 70%, who successfully received stenting. Tong‐xin‐luo group: 38 participants (age: 56.6 ± 10.9 years). Control group: 38 participants (age: 50.1 ± 12.4 years ).
Interventions	Control group: conventional treatment: • Pre‐operation(3 days before operation): aspirin 300 mg qd, clopidogrel 75 mg qd, simvastatin 20 mg qd. • Intra‐operation: heparin 10000 IU/kg, then 1000 IU/hour during operation. • Post‐operation: low molecular weight heparin 600 ml subcutaneous injection bid for 5 days; aspirin 300 mg qd, clopidogrel 75 mg qd. Tong‐xin‐luo group: conventional treatment plus Tong‐xin‐luo capsules (4 capsules, tid). Duration of intervention: 6 months. Co‐intervention: none.
Outcomes	Occurrence of angina.
Notes
Risk of bias
Bias	Authors' judgement	Support for judgement
Random sequence generation (selection bias)	Unclear risk	Randomised, but detailed information on the randomisation method was not provided.
Allocation concealment (selection bias)	Unclear risk	No description about the allocation procedure.
Blinding of participants and personnel (performance bias) All outcomes	High risk	No description about blinding. No placebo.
Blinding of outcome assessment (detection bias) All outcomes	High risk	No description about blinding. No placebo.
Incomplete outcome data (attrition bias) Angiographic restenosis	Unclear risk	The outcome was not reported.
Incomplete outcome data (attrition bias) Outcomes other than angiographic restenosis	Low risk	There was no loss to follow‐up.
Selective reporting (reporting bias)	Unclear risk	The study protocol is not available.
Other bias	Low risk	Baseline information between groups showed no statistical differences in sex, age, length of lesions, distributions of coronary pathological changes and severity of stenosis between the two groups (P > 0.05). The study appears to be free of other sources of bias.