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. 2024 Jun 24;15(6):717–729. doi: 10.5306/wjco.v15.i6.717

Figure 1.

Figure 1

Schematic representation of the interplay among dyslipidemia, atherosclerosis, and myeloproliferative neoplasms, with a focus on the main pathogenetic mechanisms that drive the development of atherosclerosis. CHIP: Clonal hematopoiesis of indeterminate potential; HSC: Hematopoietic stem cells; MPNs: Myeloproliferative neoplasms.