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. 2024 Jul 1;22:386. doi: 10.1186/s12951-024-02648-5

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© The Author(s) 2024

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 27 — (A) Schematic model depicting the effects of targeting MFG-E8 gene expression in macrophages on intestinal epithelial homeostasis. Inhibition of miR-99b stimulated the migration of intestinal epithelial cells in LPS-induced septic mice. (B) Images of the small intestine processed for BrdU/DAPI staining. (Note: green and blue colors show BrdU-labeled cells and nuclei, respectively). (C) Quantitative analysis discovered that SNA-NC anti-miR99b treatment increased enterocytes’ migration along the crypt-villus axis in LPS-induced septic mice. n = 3. **P.

permission was received from ref [165]