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. 2024 Jul 3;14:272. doi: 10.1038/s41398-024-02990-0

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© The Author(s) 2024

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 7 — A (upper panel) Digital micrographs showing confocal images of the cerebellar Crus II lobule of control (left) and VPA-exposed (right) mice. The nuclei are stained with DAPI (blue). (Lower panel) Higher magnifications of the boxed areas in (Upper panel) showing GluN2A subunit-containing NMDA receptor immunoreactivity in the GL (Green; arrows). PCL Purkinje cell layer, GCL granule cell layer. B Mean gray values of GluN2A subunit immunoreactivity in the cerebellar Crus II lobule in control and VPA-exposed mice. *P < 0.05, VPA versus control.