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. 2024 Jul 7;16(1):2374608. doi: 10.1080/19490976.2024.2374608

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© 2024 The Author(s). Published with license by Taylor & Francis Group, LLC.

This is an Open Access article distributed under the terms of the Creative Commons Attribution-NonCommercial License (http://creativecommons.org/licenses/by-nc/4.0/), which permits unrestricted non-commercial use, distribution, and reproduction in any medium, provided the original work is properly cited. The terms on which this article has been published allow the posting of the Accepted Manuscript in a repository by the author(s) or with their consent.

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Figure 9. — IAA-regulated neuroinflammation via the NF-κB signaling pathway.

(a) The TPM ratio of up-regulated and down-regulated genes. (b) The expression levels of differential expression genes in the brain detected by QPCR. (c) Analysis of protein–protein interaction networks of differential expression genes. (d) Flow chart of NF-κB signaling pathway. (e) Protein levels of DR3, p-IKK/IKK, p-IκBα/IκBα and p-NF-κB/NF-κB in the brain detected by western blot. Data are expressed as mean±SEM, *p < .05, **p < .01, ***p < .001, ****p < .0001.