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. 2024 May 21;39(5):895–907. doi: 10.1007/s11011-024-01345-4

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© The Author(s) 2024

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 5 — Overexpression of hsa_circ_0004872 blocked the interactions between EIF4A3 and PDL1 mRNA. A The correlation between hsa_circ_0004872 and EIF43A analyzed by Pearson correlation analysis. B, C The mRNA levels of EIF43A and PD-L1 in IOMM‐ Lee and CH157-MN cells with shNC or shEIF4A3 transfection were determined by RT-qPCR. D RIP assay proved the interaction between PD-L1 and EIF43A in IOMM‐ Lee and CH157-MN cells with pcDNA3.1 or pcDNA3.1-circ transfection. E RNA pull down assay demonstrated the interaction between PD-L1 and EIF43A. F The RNA stability of PD-L1 mRNA after actinomycin D was identified using RT-qPCR. *P < 0.05. **P < 0.01