Figure - PMC

Skip to main content

An official website of the United States government

Here's how you know

Here's how you know

Official websites use .gov
A .gov website belongs to an official government organization in the United States.

Secure .gov websites use HTTPS
A lock ( ) or https:// means you've safely connected to the .gov website. Share sensitive information only on official, secure websites.

View full-text article in PMC

. 2024 Jun 25;8(8):bvae126. doi: 10.1210/jendso/bvae126

Search in PMC
Search in PubMed
View in NLM Catalog
Add to search

© The Author(s) 2024. Published by Oxford University Press on behalf of the Endocrine Society.

This is an Open Access article distributed under the terms of the Creative Commons Attribution License (https://creativecommons.org/licenses/by/4.0/), which permits unrestricted reuse, distribution, and reproduction in any medium, provided the original work is properly cited. See the journal About page for additional terms.

PMC Copyright notice

Figure 1. — Adipose tissue features and distribution, and associated damage mechanisms underlying the pathophysiology of obesity. Adipose tissue undergoes several changes in its composition, distribution, and function that increase the risk of developing diabetes, cardiovascular complications, and other metabolic diseases. Unhealthy expansion of adipose tissue during obesity is mainly mediated by hyperplasia and hypertrophy of adipocytes, increasing adipose tissue inflammation driven by activation of proinflammatory adipokines and cytokines, such as IL-6 and TNF-alpha. Created with BioRender.com.