Fig. 8.

The schematic of the possible underlying mechanisms of Aβ1-42 treatment alone versus the MOL pretreatment before Aβ1-42 exposure on the prevention of apoptotic cell death in neuronal cells. Akt, Protein kinase B; Aβ, amyloid beta; Bax, B-associated X protein; Bcl-2, B-cell lymphoma-2; MOLE, Moringa oleifera leaf extract; NDUFS3, NADH dehydrogenase (ubiquinone) Fe-S protein 3; P, phosphorylation; ROS, reactive oxygen species; TIM23, translocase of the inner mitochondrial membrane 23; ΔΨm, mitochondrial membrane potential.